Protein-mediated viral latency is a novel mechanism for Merkel cell polyomavirus persistence, Proceedings of the National Academy of Sciences (2017). The researchers call this process 'protein-mediated viral latency.' However, unlike the herpes viruses, MCV has a much smaller amount of DNA and does not have its own latency mechanism. Explore further: Cancer-causing virus masters cell's replication, immortalityMore information: Hyun Jin Kwun et al. Loss of nutrients, for example, makes cells shut off some SCF E3 ligases, which allows the MCV replication protein to build up to generate new viruses.
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