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Dengue and Zika virus family uses an unexpected approach to hijack human cell machinery
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Washington, DC - Jan. 10, 2017 - A group of researchers in Germany have discovered that flaviviruses -- a family that includes Dengue, Zika and West Nile viruses -- use an unexpected mechanism to hijack the cell's machinery to replicate themselves compared to many other RNA viruses.The findings, published this week in mBio®, an open-access journal of the American Society for Microbiology, highlight new ways by which viruses manipulate human cells and may reveal new targets for designing antiviral therapies.Dengue virus and Zika virus are both positive-strand RNA flaviviruses, which means that once a virus particle infects a cell, its RNA genome can be immediately translated by cellular machinery into viral proteins to make new virus particles and spread the infection.
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Angola has recorded two cases of the Zika virus — Quartz
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Just as it seemed the Zika virus was under control around the world, Angolan health officials confirmed on Jan. 9 the country's first two cases of the disease.Africa's largest oil producer is already struggling to overcome a cholera outbreak that has infected 106 people and killed six.Last year, an outbreak of yellow fever, also a mosquito-borne virus, left at least 400 people dead, with the government only declaring an end to the epidemic in December.
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Immature form of Zika could clarify how virus infects
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Share Article FacebookTwitterEmail You are free to share this article under the Attribution 4.0 International license.Follow Futurity RSSTwitterFacebookNewsletterResearchers have determined the high-resolution structure of immature Zika virus, a step toward better understanding how the virus infects host cells and spreads.Zika belongs to a family of viruses called flaviviruses, which includes dengue, West Nile, yellow fever, Japanese encephalitis, and tick-borne encephalitic viruses.
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